Sentences with phrase «with damage to hearts»

And clinicians working with anorexics have reported that after a decade or two, those with partial or subthreshold forms of the disorder — who maintain a diet some have compared to calorie restriction — show a failure to thrive, with damage to hearts, lungs and other organs.

Not exact matches

As always, our team worked with local partners to carefully assess the damage and identify communities with the greatest need for help» noted Erik Dyson, C.E.O. of All Hands and Hearts.
Yet, I think that the Holy Spirit's heart is broken, and that the Trinity is «discussing» how best to replace that dying Pharisee - church structure with minimal collateral damage.
This is very hard to do, but it is much more damaging to live with unconfessed sin in your heart.
Eggplant is loaded with phytonutrients that act as antioxidants that help to reduce the number of free radicals in the body which damage cells and may contribute to certain diseases, including some cancers and heart diseases.
The claim is that with this diet, the damage to arteries due to heart disease can be undone.
But in my heart I can tell weaning my daughter would do so much damage, she's so attached to her «mamamilk» and I truly feel it will help her bond with her baby brother.
Comparing the heart rate or blood sugar levels of a given number of people might be beneficial in determining the range in which people maintain good health — and perhaps we can even say that by comparing children's abilities and establishing a range of «normal,» we can determine which children have difficulties and how to help them — but comparing ourselves with others, and in particular our children to other children, can have very damaging effects.
There are an estimated seven million people in the UK living with prediabetes1 and recent research has shown the condition may already be causing long - term damage to the body, especially the heart and circulatory system3.
A few years ago, the group discovered another existing compound that improved blood flow in damaged hearts, also proved to be effective in treating locally advanced or metastatic triple negative when combined with chemotherapy.
«Diseased, damaged hearts with a lot of scar tissue don't contract and relax at the same time throughout the atrium because it is more difficult to rhythmically contract the thicker, tougher damaged tissue.»
The goal of stem cell therapy is to replace the damaged tissue with new heart cells and restore the failing heart to normal function.
Scientists use mathematical modeling to simulate human mesenchymal stem cell delivery to a damaged heart and found that using one sub-set of these stem cells minimises the risks associated with this therapy.
Many children with congenital heart disease (CHD)-- the most common major birth defect in the United States — sustain brain damage that often leads to problems with behavior, thinking, and learning.
The complaint, filed in a federal district court by two prominent heart researchers, offers the first indication of just what is amiss in two papers they co-authored, which describe the heart's natural regenerative ability, and an effort to heal damaged hearts with stem cells.»
The molecular microscope's ability to more accurately diagnose rejection, before structural damage has occurred in a patient's heart, provides us with an essential tool in the evolution towards true precision medicine.»
The study looked at 7,470 patients with PAD who took part in the COMPASS study, and found the combination reduced heart attacks, stroke or cardiovascular death by 28 per cent and damage to limbs by 46 percent.
Now, with new kinds of technologies that are coming up, new types of tissue engineering and, you know, some of the hopes that people have for stem cells and [the] like, it may be interesting to see if there are other ways, alternatives to dealing with really badly damaged hearts that would involve growing a new heart or replacing or repairing the damage d to a badly damaged heart that might make artificial hearts less important in the somewhat more distant future.
These conditions are associated with high blood sugar, which can damage the vessels that supply blood to vital organs and can also increase the risk of heart disease and stroke.
In the United States, about 720,000 residents experience a heart attack annually, which means that hundreds of thousands of heart patients are living with the disabling complications of heart disease who could benefit from therapies to repair and regenerate damaged heart tissue.
The day after his disciplinary dismissal from University of Tokyo for «damaging the university's honor or credibility,» Hisashi Moriguchi maintained in an interview with ScienceInsider that he really did participate in a groundbreaking experiment to treat a heart disease patient with cardiac muscle cells derived from the patient's own induced pluripotent stem (iPS) cells.
In the Rutgers study, Zong and lead author Ji - An Pan, a scientist in his laboratory, looked at liver and heart damage in laboratory mice and found that the mice in which the TRIM21 gene was inactivated suffered little heart or liver damage when put through the same laboratory procedures used to produce tissue damage in mice with the gene.
Most importantly, these studies show that treatment with vitamin D3 can significantly restore the damage to the cardiovascular system caused by several diseases, including hypertension, atherosclerosis, and diabetes, while also reducing the risk of heart attack.
In research published in Molecular Cell, Rutgers scientists discovered that a protein (p62), which is supposed to act as an antioxidant to prevent cell damage, was not working efficiently in laboratory mice with liver and heart disease that mimicked these conditions in humans.
According to Steven Tsui, who is director of mechanical assist heart services at Papworth Hospital in Cambridge, and will run the UK trials, the VentrAssist is less likely to damage red blood cells because it moves the blood more slowly with a bigger impeller.
A blood test for a protein called high - sensitivity troponin T, which is released into the bloodstream when injury to the heart occurs, can identify patients with heart damage after non-cardiac surgery whose lives could potentially be saved with timely treatment, according to research presented at the American College of Cardiology's 66th Annual Scientific Session.
New research at Rockefeller University and Columbia University suggests these small molecules may be able to relay valuable information about damage to the heart: Scientists in Thomas Tuschl's Laboratory of RNA Molecular Biology have linked an increase in certain microRNAs circulating in the blood with injury to cardiac muscle.
Struggling to balance on one leg for 20 seconds or longer was linked to an increased risk for small blood vessel damage in the brain and reduced cognitive function in otherwise healthy people with no clinical symptoms, according to new research in the American Heart Association's journal Stroke.
Researchers have even begun to test whether hydrogen sulfide can protect us against the damage associated with heart attacks and strokes (Science, 30 May 2008, p. 1155).
If the infection isn't stopped, it can progress into rheumatic fever — a highly contagious disease that affects more than 400,000 people worldwide each year and leaves survivors with lasting damage to heart valves and other tissue.
Rutgers scientists have discovered that a protein which is supposed to prevent cell damage is not working efficiently in laboratory mice with heart and liver disease.
At the same time, when mice that lacked the RGS6 protein were treated chronically with alcohol, they experienced less damage to heart and liver as well as the lining of the gut compared to mice with the protein.
«To our knowledge RGS6 is the only gene with a demonstrated ability to promote alcohol - seeking behaviors while simultaneously worsening the damaging effects of alcohol consumption on the heart, stomach, intestine and liver,» Fisher sayTo our knowledge RGS6 is the only gene with a demonstrated ability to promote alcohol - seeking behaviors while simultaneously worsening the damaging effects of alcohol consumption on the heart, stomach, intestine and liver,» Fisher sayto promote alcohol - seeking behaviors while simultaneously worsening the damaging effects of alcohol consumption on the heart, stomach, intestine and liver,» Fisher says.
With our new technology, we can quickly create billions of these cells in a dish and then transplant them into damaged hearts to treat heart failure.»
Nanoparticle «bombs» to kill cancer, molecular - sized bridges to repair damaged hearts, and scarless surgery techniques are now on the frontier of medical innovations, with the new institute leading the way.
In mice with heart damage similar to a heart attack in humans, the three factors not only created new muscle, but also improved the pumping of the heart.
Yin noted that with a better understanding of the genetic underpinnings that lead to tissue regeneration, he and his team may be able to develop new drug candidates that control the activity of the genetic circuits and repair damaged hearts.
Friedreich's ataxiais an inherited disease that causes progressive damage to the nervous system, resulting in symptoms ranging from gait disturbance to speech problems; it can also lead to heart disease and diabetes.The ataxia of Friedreich's ataxia results from the degeneration of nerve tissue in the spinal cord, in particular sensory neurons essential (through connections with the cerebellum) for directing muscle movement of the arms and legs.
With the loss of the cushioning effect provided by free - moving collagen in the blood vessels, the force of the surge of blood that is driven into the arteries by the pumping action of the heart is carried directly to organs like the kidneys and the brain, damaging to the structures that filter our blood and that connect the functional regions of our brain, and putting us at risk of a stroke.
The three factors, Gata4, Mef2c, and Tbx5 (GMT), work together to turn heart genes on in these cells and turn other genes off, effectively regenerating a damaged heart with its own cells.
But heart cells never truly regenerate in the damaged tissue, and myofibroblasts compensate for their absence by forming a stiff, collagenous scar that interferes with the heart's ability to maintain stable heart rhythms and to expand and contract forcefully to pump blood.
The potential to create new muscle cells through cell division, much like a salamander does, could offer new hope to the millions living with damaged hearts.
Dorr had no idea the laboratory he envisioned would one day bring science to the brink of the conquest of aging - related diseases, lead to the development of drugs with the potential to repair damaged hearts or grow to become an international center for training in the biomedical sciences.
But the damage caused by a heart attack can result in heart failure — and patients diagnosed with heart failure have an average life expectancy of less than five years, according to Dr. Chuck Murry, director of the Center for Cardiovascular Biology at the University of Washington in Seattle.
«Our hope is to one day treat diseases like heart failure or Parkinson's disease with drugs that help the heart and brain regenerate damaged areas from their own existing tissue cells.»
While the scar limits further tissue damage in the short term, over time its stiffness interferes with the heart's ability to pump, leading to disability and ultimately to terminal heart failure.
The findings may lead to improved regenerative therapy for people with heart damage.
«These findings could have a significant impact on patients with heart failure, whose damaged hearts make it difficult for them to engage in normal activities like walking up a flight of stairs,» said Qian.
WEDNESDAY, Oct. 24 (HealthDay News)-- In a surprising finding, people with sleep apnea appear to suffer less heart damage during a nonfatal heart attack than those without the condition, researchers report.
As explained by cardiologist Dr. Alfred Bove, a STEMI heart attack is characterized by certain readings on an electrocardiogram, and «is usually associated with pretty significant damage to the heart muscle.»
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