The problem with using meters designed for humans is that they were designed to work
with human red blood cells which are larger than dog and cat red blood cells.
Not exact matches
First x object was created out of nothing, then combined
with other things created out of nothing, then magically an atom, yhen a
cell, a molecule, then bacteria, single
cell creatures, followed by simple sea creatures
with organs, then more advanced creatures, next
red blooded mammals, then primates, and finally
human.
Their major hurdle: to come up
with a replacement for hemoglobin (an iron - enriched protein in
red blood cells that transports oxygen from the lungs to the rest of the body) that can be directly introduced into the
human circulatory system.
The researchers created the nanosponges by separating the membranes of
human red blood cells from their internal contents and stabilizing the membranes
with an engineered core designed to absorb the toxins produced by pathogenic bacteria.
Human red blood cells are usually disc - shaped
with a central dimple but conditions such as sickle -
cell anaemia alter their shape.
Researchers have discovered that protection from the most severe form of malaria is linked
with natural variation in
human red blood cell genes.
These mice have their hemoglobin genes removed and replaced
with the mutated
human version, saddling them
with many of the same problems as
human sufferers, including immature, short - lived, and sickle - shaped
red blood cells; anemia; reduced
blood flow; and an enlarged spleen.
«What makes it particularly interesting is that the region we can show is associated
with protection happens to be right up against a set of genes we know are related to how malaria invades the
red blood cell,» study author Dominic Kwiatkowski of the Wellcome Trust Sanger Institute and the Wellcome Trust Centre for
Human Genetics told The Post.
By examining the results of genome - wide association studies (GWAS) in conjunction
with experiments on mouse and
human red blood cells (RBCs), researchers in the lab of Whitehead Institute Founding Member Harvey Lodish have identified the protein cyclin D3 as regulating the number of
cell divisions RBC progenitors undergo, which ultimately affects the resulting size and quantity of RBCs.