While no evidence in support of our hypothesis d was found, hypothesis c was therefore supported. (link.springer.com)
In short, the results supported neither of our hypotheses. (academic.oup.com)
Clinical evidence in support of this hypothesis comes from a number of XP complementation group D patients that do not fit within the framework of the current monoallelic paradigm of XPD disorders (Figure 5). (journals.plos.org)