The term
"allergic airway inflammation" refers to a condition in which the airways (such as the bronchi and lungs) become swollen, irritated or constricted due to an allergic reaction.
Full definition
To relieve the symptoms of patients with allergic asthma, the treatment
of allergic airway inflammation with glucocorticoids and the use of a bronchodilator asthma spray will continue to be paramount in the near future.
Previous studies have demonstrated that there are various levels of inflammatory responses in patients who suffer from asthma, and
allergic airway inflammation with increasing EOS is the main pathological feature of asthma (20,21).
«Existing therapies for airway obstructive diseases provide relief by
preventing allergic airway inflammation, but none of these drugs specifically address the problem of excessive mucus production; further, there is no convincing evidence that current therapies significantly reduce mortality associated with chronic asthma and COPD,» Srivastava said.
He did his Ph.D from University of Pune in Biotechnology in identifying posttranscriptional regulation of cytokine IL - 10 and its effect
on allergic airway inflammation.
Tobias Polte and his team from the Department of Environmental Immunology at the Helmholtz Centre for Environmental Research (UFZ) collaborated with Jan Simon and his colleagues from the Clinic for Dermatology, Venereology and Allergology at the University of Leipzig and have recently been successful in discovering a molecule that plays a significant role in the development
of allergic airway inflammation — as demonstrated in their recent publication in Nature Communications.
UFZ researchers and their colleagues from the University of Leipzig have recently been successful in finding a protein that plays a critical role in the development of
allergic airway inflammation.
Dr. Julie Deckers (VIB - UGent): «As predicted, our test showed that house dust mite - induced skin inflammation leads to aggravated levels of
allergic airway inflammation.
Enhanced Expression of Myristoylated Alanine — Rich C Kinase Substrate (MARCKS) in Airway Epithelium of an Animal Model of
Allergic Airway Inflammation and Human Patients with COPD