And according to a new study, you should hope you're endowed with ample stretches of
DNA called telomeres at the ends of your chromosomes.
With each division, the stretches of DNA at the ends of their chromosomes —
regions called telomeres — begin to fray and shorten, leaving the remaining DNA more liable to errors and mutations.
Telomeres are composed of double - stranded DNA with terminal 3» single - stranded G - rich
overhangs called telomere G - tails.
As it turns out, a certain gene,
called telomere repeat binding factor 2 (Terf2), belongs to a complex of six telomere - associated proteins, termed shelterin.
Without its activity, protective chromosomal
caps called telomeres would gradually shorten with each cell division.
Researchers have suspected for about 12 years that human cell division is regulated by
structures called telomeres, specialized stretches of DNA located at the ends of the chromosomes.
Telomerase is an enzyme that replicates the ends of chromosomes (sections of
DNA called telomeres), replacing the DNA lost when chromosomes are copied before cell division and, therefore, maintaining the stability of the genome.
Scientists believe that the reason for these amazing benefits is due to
something called telomeres, which are the caps at the end of each strand of DNA that protect our chromosomes.
The team analysed their DNA, as well as other signs of ageing, such as insulin resistance — which is linked to diabetes — and the length of the caps on the ends of their chromosomes,
called telomeres.
Shorter chromosome caps,
called telomeres, have been linked to increased risk of death.
Our DNA comes equipped with a stretch of nonsense or «noise» DNA
called the telomere.
These tiny strips of DNA,
called telomeres, cap the end of chromosomes.
Normally, cell division halts once these structures,
called telomeres, wear down.
DNA damage, including the shortening of chromosome endcaps (
called telomeres) is also a prime suspect.
The «caps» Eisenberg and his co-authors measured are
called telomeres.
The ends of chromosomes are capped by wads of DNA
called telomeres.
The myth has a kernel of truth, because the ends of chromosomes are protected by specialized stretches of DNA
called telomeres.
There are various causes of senescence: Damage to the DNA is just as possible as is the attainment of a maximum number of divisions (limited by the so -
called telomeres).
Normal cells lose a little bit of their DNA every time they reproduce as the molecule's strands lose part of their protective tips,
called telomeres.
I know it sounds like a late - night TV offer, with a vegetable dicer thrown in for free, but one of the most exciting areas of aging research is on the importance of the length of the tips of your chromosomes,
called telomeres.
Our chromosomes have little «caps» on
them called telomeres, and a short telomere is basically a sign of age.
They're
called telomeres.
Aging begins at the cellular level, at the level of tiny structures
called telomeres that protect the ends of chromosomes when cells divide.
Work - related stress shortens protein camps at the ends of chromosomes,
called telomeres.
Our genes are packaged into structures called chromosomes and at the tips of these chromosomes are structures
called telomeres.
That cap is
called a telomere.