Sentences with phrase «low density lipoprotein»

The high - fat DASH significantly lowered triglycerides, lowered the size of very low density lipoprotein (VLDL) and raised LDL particle size, larger size LDL lowers risk.

The results support reduction in low density lipoprotein cholesterol as the primary goal for lipid modifying interventions.»

Alfalfa seeds lower low density lipoprotein cholesterol and apolipoprotein B concentrations in patients with type II hyperlipoproteinemia.

Protective Effect of Irisin on Atherosclerosis via Suppressing Oxidized Low Density Lipoprotein Induced Vascular Inflammation and Endothelial Dysfunction.

It reduces low density lipoproteins (LDL), total cholesterol, and lowers your risk of heart disease.

Harmful low density lipoprotein (LDL) cholesterol and stored fat in the form of triglycerides are both reduced (1).

Soluble fiber also had been found to help lower serum cholesterol levels, particularly undesirable low density lipoprotein (LDL) cholesterol.

Chemist Harold Schmitz of the M&M Mars candy company took the work a step further, showing that a family of flavonoids, a subclass of chocolate polyphenols, helped neutralize low density lipoprotein or «bad cholesterol» in the test tube.

A complementary DNA encoding the human low density lipoprotein (LDL) receptor under control of the mouse metallothionein - I promoter was injected into fertilized mouse eggs, and a strain of mice expressing high levels of LDL receptors was established.

Protein Phosphatase 2A (PP2A) Regulates Low Density Lipoprotein Uptake through Regulating Sterol Response Element - binding Protein - 2 (SREBP - 2) DNA Binding.

Mechanisms of hepatic very low density lipoprotein overproduction in insulin resistance.

«While the major CVD benefit of statins is due to reduction in plasma low density lipoprotein cholesterol (LDL - C), [2] statins also produce moderate increases, ranging from 4 % to 10 %, in levels of high density lipoprotein cholesterol (HDL - C).

Black tea theaflavins and flavonoids help manage atherosclerosis, 7 aids in maintaining healthy cholesterol levels, 1 and supporting endothelial operations resulting in healthy circulation and integrity, 2,4,5 and helps control oxidative damage.3 A special extract of Indian gooseberry (Capros amla) contains beneficial tannins that may improve endothelial actions and supports low density lipoprotein (LDL), high density lipoprotein (HDL), and triglycerides.6

The «bad» cholesterol is LDL or low density lipoproteins while mainstream medicine considers HDL (high density lipoprotein) benign or even «good» cholesterol.

Because saturated fats have no gaps (or double bonds) they are able to pack together really tightly so when they enter the bloodstream, they tend to raise low density lipoprotein (LDL) or bad cholesterol.

Coconut oil, therefore, does not produce any significant change in the circulating VLDL [very low density lipoprotein], which is supposed to be bad for vessel thickening.

Changes in plasma very low density and low density lipoprotein content, composition, and size after a fatty meal in normo - and hypertriglyceridemic man

«Statin drug trials have provided the major evidence for the benefits of a therapy that lowers low density lipoprotein (LDL), the so called «bad cholesterol,» and statin therapy is now the mainstay of clinical management of cardiovascular disease.

Lipoprotein lipid profile disturbances associated with an elevated TG level include low High Density Lipoprotein (HDL) cholesterol and HDL particle (HDL - P), and a predominance of small dense Low Density Lipoprotein (LDL) and LDL particle (LDL - P).

Phytosterols, on the other hand, are plant sterols that compete with and decrease low density lipoproteins (LDL's) while not lowering the good high density lipoproteins (HDL's).

LDL stands for Low Density Lipoprotein and HDL stands for High Density Lipoprotein.

The high fiber group also reduced their total cholesterol by nearly 7 %, their triglyceride levels by 10.2 % and their VLDL (Very Low Density Lipoprotein — the most dangerous form of cholesterol) by 12.5 %.

In serum and tissues, very low density lipoprotein (VLDL) cholesterol levels werelowered and HDL - cholesterol was increased.

Lipid concentrations (very low density lipoprotein, low density lipoprotein, high - density lipoprotein - 2, high - density lipoprotein - 3, apoprotein B, and total triglycerides) were not different at nine months among infants exclusively breastfed for nine months versus those exclusively breastfed for six months and mixed breastfed from six to nine months.

Breast fed OR Breast feed OR Breast feeds OR Breast feeding OR Breast milk OR Bottle fed OR Bottle feed OR Bottle feeds OR Bottle feeding OR Infant feed OR Infant feeds OR Infant feeding OR Infant nutrition OR Formula fed OR Formula feed OR Formula feeds OR Formula feeding OR Infant diet OR Dried milk OR Early nutrition AND Blood cholesterol OR Serum cholesterol OR Blood total cholesterol OR Serum total cholesterol OR Blood lipid OR Serum lipid OR Low density lipoprotein

The current studies were designed to determine whether chronic overexpression of low density lipoprotein (LDL) receptors in the liver would protect mice from the increase in plasma LDL - cholesterol that is induced by high - fat diets.

Overexpression of low density lipoprotein (LDL) receptor eliminates LDL from plasma in transgenic mice

Dr. Brown and his colleague, Dr. Joseph L. Goldstein, discovered the low density lipoprotein (LDL) receptor, which controls cholesterol in blood and in cells.

Discovered that apoE is a major ligand for the low density lipoprotein receptor by mapping the amino acid residues involved in receptor binding and defining the role of apoE in liver clearance of lipoproteins.

Dr. Goldstein and his colleague, Michael S. Brown, discovered the low density lipoprotein (LDL) receptor and worked out how these receptors control cholesterol homeostasis.

Structural model of the PCSK9 protein that mediates degradation of the low density lipoprotein (LDL) receptors.

He and his long - time colleague, Dr. Joseph L. Goldstein, together discovered the low density lipoprotein (LDL) receptor, which controls the level of cholesterol in blood and in cells.

Trans fatty acids tend to raise the level of low density lipoproteins (LDLs = bad) and lower the level of high density lipoproteins (HDLs = good).