Sentences with phrase «of obese mice»

The study shows that a hormone acting on the placenta can protect the offspring of obese mice from being born overweight.

Colonies of those bacteria chowed down and grew, taking over the guts of obese mice and crowding out bad actors, like pro-inflammatory bacteria.

The taste tissues of the obese mice had a higher amount of a type of protein called a cytokine, which regulates inflammation, than their normal - weight kin, the researchers found.

«We did not do it using a genetic strain of obese mice, but mice that became obese the way that people do, by eating a high - fat, high - sugar diet.»

Now, a new study of obese mice reveals that whole - body vibration provides similar metabolic benefits as walking on a treadmill, suggesting it may be useful for treating obesity and type II diabetes.

Consistently, killing the intestinal bacteria of obese mice with an antibiotic treatment reduced body fat and improved insulin sensitivity (4, 5).

Studies found that transplanting the gut bacteria of obese mice into lean mice caused the lean mice to gain fat cells quickly.

A second study fed macadamia nuts or a placebo to two groups of obese mice.

In the case of the obese mice, Sinclair observes, resveratrol increased insulin levels while decreasing glucose levels, resulting in healthier liver and heart tissue when compared with obese mice that did not receive treatment.

The large amounts of fat around the testes of obese mice, «could alter the environment and encourage epigenetic changes», says Teague, who presented the results at the 14th World Congress on Human Reproduction in Melbourne, Australia, this month.

The mice that received the microbiomes of the obese mice gained significantly more weight than did the mice with the lean - mouse microbiomes.

«The virus penetrates more deeply into the lungs of obese mice, and the animals seem to have a more difficult time repairing the damage,» Schultz - Cherry said.

The investigators also discovered that a potent suppressor of autophagy (called mTOR) was hyperactivated in the kidneys of obese mice, and treatment with an mTOR inhibitor ameliorated autophagy insufficiency.

In research published in December 2013, the investigators found that high levels of IKK - ε and TBK1 meant that certain receptors in the fat cells of obese mice were unable to respond to neurotransmitters called catecholamines, which are generated by the sympathetic nervous system and promote «fat - burning.»

In their research the team blocked a particular inflammatory signalling pathway (IKKβ / NF - κB) in the brains of obese mice.

Dr Tups says the team found that administering butein either directly into the brain or orally greatly improved glucose tolerance and brain insulin signalling in both types of obese mice.

Numerous animal studies suggesting STACs can extend lifespans of obese mice and counter age - related diseases have been published.

The hormone reversed the abnormally high fetal blood sugar levels observed in the fetuses of obese mice, and it seemed to limit fetal nutrition by reducing levels of certain glucose and amino acid transport proteins at the placental barrier.

«Based on our composite data,» explains Dr. Gendler, «it appears that methotrexate or cyclophosphamide can induce the livers of obese mice to burn off rather than accumulate excessive dietary fat.

Surprisingly, the effects of obesity on gut bacteria, inflammation, and osteoarthritis were completely prevented when the high fat diet of obese mice was supplemented with a common prebiotic, called oligofructose.

While a common prebiotic supplement did not help the mice shed weight, it completely reversed the other symptoms, making the guts and joints of obese mice indistinguishable from lean mice.

The knee cartilage of obese mice who ate the oligofructose supplement was indistinguishable from that of the lean mice.

This basically tells us that certain members of the Bacteroidetes family seem to be more aggressive when compared to different members of the microbiota of obese mice.

They found that 75 percent of the obese mice remained cancer - free, while 33 percent of the non-obese did.

Earlier studies in overfed mice in gestation and lactation periods demonstrated IGT in male and female offspring of obese mice.

Cinti S, Mitchell G, Barbatelli G, Murano I, Ceresi E, Faloia E, Wang S, Fortier M, Greenberg AS and Obin MS. «Adipocyte death defines macrophage localization and function in adipose tissue of obese mice and humans.»

Francis Benedict reported this in 1936, when he fasted a strain of obese mice.

In a study of obese mice that received a diet rich in either coconut or safflower oil, mice that received the coconut oil were found to have made much less fat cells than mice that received the safflower oil.

«In studies conducted on mice, the guts of obese mice had higher levels of firmicutes and lower levels of bacteroidetes.

A common antioxidant found in human breast milk and foods like kiwi fruit can protect against nonalcoholic fatty liver disease (NAFLD) in the offspring of obese mice, according to researchers at the University of Colorado Anschutz Medical Campus.

While hardly any inflammation occurred in the subcutaneous fat of obese mice and cGMP signaling was largely intact, things were very different for the deeper - lying abdominal fat: through the significant weight increase, inflammation had spread and the fat - burning turbocharger cGMP largely came to a standstill.

One group consisted of lean mice without having been tampered, another group of lean mice had their myostatin blocked, the third group consisted of obese mice that had a normal myostatin level and the final was made out of obese mice with blocked myostatin.

This study showed that the microbiota of lean mice (one group of mice was lean and the other was obese) was more invasive than those of obese mice.