Sentences with word «myogenesis»

The myoD family of DNA binding proteins has been implicated in the control of myogenesis in a variety of organisms.

However, Setd7 expression increased with the progression of myogenesis in vitro indicating the potential importance of this event for skeletal muscle development and regeneration in vivo.

In the past years, we focused our attention on Srf transcription factor which is one of the three master genes that controls myogenesis in Caenorhabditis elegans, together with MyoD and HAND.

Thus, we identified, through a genome - wide analysis, two early developmental molecular involved both in myogenesis as well as in neurite formation and establishment of neuromuscular connections.

In this study, Chazaud et al., evidenced that endothelial cells (ECs) and myogenic progenitor cells (MPCs) interacted to couple myogenesis and angiogenesis during skeletal muscle regeneration.

«For several years, we have been studying myogenesis, a process by which muscles are formed during embryonic development,» says Jean - François Côté, PhD, Director of the Cytoskeletal Organization and Cell Migration research unit at the IRCM.

To assess the role of hlh - 1 in C. elegans myogenesis, genetic deficiencies spanning the hlh - 1 locus were isolated after gamma irradiation.

A defective Krab - domain zinc - finger transcription factor contributes to altered myogenesis in myotonic dystrophy type 1.

Preliminary data we have now obtained further indicate that in somite cells, the activation of the NOTCH pathway triggers a «signalling module» that couples the initiation of myogenesis with the epithelial - mesenchymal transition (EMT) that allows them to migrate into the growing muscle.

Cytoplasmic NOTCH and membrane derived β - catenin link fate choice to epithelial - mesenchymal transition during myogenesis

Coupling between Myogenesis and Angiogenesis during Skeletal Muscle Regeneration Is Stimulated by Restorative Macrophages

Accordingly, our data suggests that the failure of regenerative myogenesis to keep pace with disease progression in DMD is not due to muscle stem cell exhaustion, but rather is due to a cell - autonomous deficiency in asymmetric division.

A recent study from the laboratories of Thomas A. Rando (Stanford University, California, USA) and Fabio M. Rossi (University of British Columbia, Canada) now provides evidence that modulation of Setd7, a lysine methyltransferase previously indicated to play a role in myogenesis [2], can enhance the therapeutic value of in vitro cultured MuSCs [3] and may represent the way forward for MuSC - based therapies.

Moreover, we showed that via the production of Oncostatin - M, restorative macrophages promoted myogenesis / angiogenesis coupling.

Senesi P, Luzi L, Montesano A, Terruzzi I. DNA demethylation enhances myoblasts hypertrophy during the late phase of myogenesis activating the IGF - I pathway.

Jesty SA, Steffey MA, Lee FK, et al. ckit + precursors support postinfarction myogenesis in the neonatal, but not adult, heart.

Miostatin (also known as growth differentiation factor 8 or GDF - 8), is a myokine produced and released by myocytes that inhibits myogenesis, i.e. growth and differentiation in muscle cells.

In the course of this process, signals from the microenvironment instruct cycling MuSCs and control myogenesis.

We have recently shown that in avian embryos, muscle formation is initiated by Delta1 - positive neural crest cells migrating from the dorsal neural tube that, in passing, trigger NOTCH signalling and myogenesis in selected epithelial somite progenitor cells, allowing them to migrate into the nascent muscle to differentiate.

In chick embryos, skeletal muscle formation is initiated by migrating Delta1 - expressing neural crest cells that trigger NOTCH signaling and myogenesis in selected epithelial somite progenitor cells, which rapidly translocate into the nascent muscle to differentiate.

EC - and MPC - derived Apelin, Oncostatin - M and Periostin controlled myogenesis / angiogenesis coupling and were required for myogenesis and vessel formation.

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