When they are taken, the cancer cells eat the maple syrup because cancer cells consume 15 times more
glucose than normal cells.
«What we're starting to learn is that there can be bad cells from cancer that appear to benefit more from
antioxidants than normal cells,» he said in an interview.
By reviewing the experimental studies that compared the mechanics of individual normal and cancer cells, we argue that cancer cells can indeed be considered as
softer than normal cells.
Cancer cells use much more
sugar than normal cells, and they do it by changing the way they use these energy sources from the environment.
Cancer cells are known to have anywhere from ten to thirty times more insulin
receptors than normal cells and depend upon anaerobic metabolism of sugar for fuel.
So, even though a cancer cell has far more
glucose than a normal cell, less of this glucose gets inside its mitochondria than in a normal cell.
«Tumor cells produce larger quantities of H2O2 and use oxidative signals at higher
levels than normal cells in order to drive their own growth,» says Mirko Sobotta, first author of the publication.
Sixth, ellagic acid (i.e. from ellagitannins), is combined with glucose, making sure that cancer cells get a larger dose of ellagic acid
than normal cells because cancer cells consume about 15 times more glucose than other cells.
In addition, cells under stress often behave
differently than normal cells do, a huge stumbling block for scientists trying to draw connections between their experiments and the natural world.
Our data suggest the reason for this: cancer cells benefit more from antioxidants
than normal cells do.»
«Some of these high - temperature organisms have salt concentrations 10 to 20 times
higher than normal cells,» says Slesarev.
In experiments with mice, the researchers found that Paneth cells engineered to lack a functional ATG16L1 gene were five times more likely to die in the face of rising TNF - alpha
signals than normal cells.
Fat cells cultured from the body mass index of a morbidly obese patient cause multiple myeloma cells to anchor to a much greater
extent than normal cells and produce a significantly larger number of blood vessels to sustain the cancer cells.
And because mouse embryo cells with inactivated copies of BRCA2 are more sensitive to ionizing
radiation than normal cells are, «it's a reasonable extrapolation» that breast cancers with mutated copies of the gene may be especially good candidates for radiation therapy.
The researchers discovered that the tumour cells had many more
mutations than normal cells, and that not only was each bowel cancer genetically different, but each cell they had studied within that cancer was different.
«One of the characteristics of cancer cells is that they tend to have more oxidative
stress than normal cells,» said Bret Freudenthal, Ph.D., lead author of the paper and postdoctoral fellow in Wilson's group.
Then the researchers studied these «iPSC - hepatic cells» and found the diseased cells secrete AAT protein more
slowly than normal cells.
«From a therapeutic standpoint you could imagine that the cells that are addicted to these nutrients could be starved and killed more
easily than normal cells.»
They believe the mutated gene causes cancer by triggering cytoskeleton malfunction, which allows the cancer cells to move more
quickly than normal cells, essentially invading the surrounding healthy tissue.
«Since cancer cells take up more
FDG than normal cells, the PET scanner can construct a 3 - D map of the cancer site by detecting the signal from the FDG.
1 — Direct Cancer Cell Death: The principle of hyperthermia is that cancer cells are much more sensitive to and intolerant of the effects of excessive
heat than normal cells.
This two - step strategy would starve cancer cells — which tend to require much higher amounts of
energy than normal cells — by limiting their two major pathways for ATP production.
«Prostate cancer cells, as well as some other solid tumors, have been shown to contain higher cholesterol
levels than normal cells,» said senior author Donald McDonnell, Ph.D., chairman of the Department of Pharmacology and Cancer Biology at Duke.
Cancer cells rely on this signal pathway more
than normal cells because their DNA is more jumbled and prone to becoming tangled.
A team of scientists at the Children's Research Institute at UT Southwestern (CRI) has made a discovery that suggests cancer cells benefit more from
antioxidants than normal cells, raising concerns about the use of dietary antioxidants by patients with cancer.
The liver releases the glucose and cancer cells are likely to pick up this glucose because cancer cells consume about 15 times more glucose
than normal cells.