Sentences with phrase «young mice»

To examine its effects, the researchers cut calorie intake by 40 percent in young mice and by 30 percent in middle - aged monkeys.

In 2012, researchers reversed some of the effects of cognitive decline by pumping the blood of young mice into old mice.

The authors went on to study young mice with white - matter nerve damage similar to that seen in premature babies.

Three new studies suggest compounds in the blood from young mice improve an older animal's ability to learn, remember and build new muscle.

After an injection of the hormone, old mice could run as far as younger mice.

But, she notes, the stem cells were collected from very young mice.

Previously they found that in addition to old mice benefiting from young blood, young mice exposed to old blood suffered memory declines, suggesting old blood may contain «aging» factors.

They found that while treatment improved young mice, it did not make a noticeable difference in the older mice.

The effect can work both ways — young mouse stem cells lose potency in old blood.

After exposure to cell - stressing x-rays or chemotherapy drugs, young mice went gray in an unexpected way.

And supplying young mice with certain sugars typically provided in breast milk helps to make sure the right microbial community gets established.

For example, laboratory experiments show that young mice stay close by their mothers for safety.

In young mice, the bones also grew slower than normal.

The team also joined together 12 pairs of old mice and 10 pairs of young mice, as controls.

After receiving an injection of neural stem cells from young mice, however, they performed as well as healthy mice did.

These mice were able to control the infection as efficiently as young mice.

Coates and colleagues focused on the initial immune response to the flu, using healthy adult and young mice who have not had previous exposures to the virus.

In their research on young mice, for instance, all the subjects died when they were infected with the Nodamura virus, but when Ding's researchers removed the suppressor protein called B2 from the virus, the infected mice began producing huge armies of the virus - attacking siRNAs and lived, unaffected by the otherwise lethal infection.

But news accounts mostly ignored the flip side of the experiment: Being tethered to an old mouse made young mice age faster.

The scientists, particularly Sedigheh Delmaghani, studied young mice whose pejvakin gene had been inactivated.

To test the effect of VCAM1, Yousef injected young mice with blood plasma taken from older mice.

After having verified that theoretical predictions and experiments matched, IBS scientists applied the technique to a 340 μm - thick young mouse skull.

Researchers at the University of Southern California have developed a prostate cancer vaccine that prevented cancer in 90 percent of young mice genetically predestined to develop the disease.

They found that when young mice are learning, a molecular fragment known as an acetyl group «caps» a particular site on the histone protein that DNA wraps itself around.

They began by adding one of three commonly used sweeteners — saccharin, sucralose or aspartame — to the drinking water of healthy young mice.

As expected, young mice treated with THC performed slightly worse on behavioral tests of memory and learning.

Part of the problem, he says, is that the incidence of many human chronic diseases rises with age, yet many researchers prefer using young mice because of the pressures of being published and getting funding.

When the scientists removed patches of skin, the older ones lacking telomerase healed much more slowly than old mice with normal telomerase or young mice lacking telomerase.

While young mice (18 weeks, equivalent to young adults) lost only a few percent of body weight on the starvation low protein diets, elderly mice (2 years old) wasted away on low protein diets.

The researchers also revealed that loss of Lsd1 in younger mice resulted in premature fat cell transformation.

The landmark 2013 study by Dr. Sinclair demonstrated that supplementation with NMN increased levels of NAD + and reversed age related degeneration in mice, giving older mice the muscle capacity, endurance and metabolism of much younger mice — the «equivalent of a human 60 year old becoming more like a 20 year old» (24).

Our answer to this question is Quill, a confident, secure, self - reliant, impetuous young mouse.

For example, after receiving THC, young mice took longer to learn where a safe platform was hidden in a water maze, and they had a harder time recognizing another mouse to which they had previously been exposed.

Yang's group took advantage of genetically engineered young mice that have protein markers which glow in response to changes in nerve function.

«This well - designed set of experiments shows that chronic THC pretreatment appears to restore a significant level of diminished cognitive performance in older mice, while corroborating the opposite effect among young mice,» wrote Susan Weiss, director of the Division of Extramural Research at the National Institute on Drug Abuse, who was not involved in the study, in an e-mail.

In October researchers at Columbia University reported that young mice predisposed to acquiring Alzheimer's accumulate protein clusters in synaptic mitochondria and that these clusters directly impair synapse function.

«It just reeks of snake oil,» said Michael Conboy, a cell and molecular biologist at the University of California, Berkeley, who's collaborated on studies sewing old and young mice together and transfusing blood between them.

Reconstructions of blood vessels in an old mouse's brain (left) and in an old mouse that received young mouse blood.

As the team reports online this week in Proceedings of the National Academy of Sciences, the homozygous mice didn't make the ultrasonic sounds that young mice emit when separated from their mothers.

Cellular - level changes to a part of the brain's reward system induced by chronic exposure to the psychoactive component of marijuana may contribute to the drug's pleasurable and potentially addictive qualities, suggests a study in young mice published in JNeurosci.

Petris found that young mice missing the ATP7A gene in their intestinal cells were unable to absorb copper from food, resulting in an overall copper deficiency that mimics symptoms of Menkes disease in children.

If an old mouse was p16 - deficient, it regrew pancreatic cells and cured itself like a normal young mouse.